Effects of non-insulin dependent diabetes mellitus on the reactivity of human internal mammary artery and human saphenous vein
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2017, Vascular PharmacologyCitation Excerpt :This study demonstrated that both NO-sensitive sGC-mediated relaxation by nitroglycerin and NO-insensitive sGC-mediated relaxation by BAY 60-2770 are preserved in internal thoracic arteries obtained from type 2 diabetic patients, which is in line with a report showing no influence of type 2 diabetes on the arterial NO-sensitive and -insensitive sGC activities [13]. Additionally, many researchers have confirmed that the relaxant response to an NO donor is comparable between non-diabetic and diabetic internal thoracic arteries [22–24]. Therefore, the presence of type 2 diabetes might not have a large influence on vascular sGC redox homeostasis.
Bilateral internal thoracic artery harvest and deep sternal wound infection in diabetic patients
2013, Annals of Thoracic SurgeryCitation Excerpt :Coronary artery bypass grafting is superior to percutaneous intervention in diabetic patients with multivessel disease [26], with the ITA providing the incremental benefit [27]. The innate resistance of the ITA to atherosclerosis, intimal hyperplasia, and medial calcification [28] persists in diabetic patients, who demonstrate a tendency toward increased vasoreactivity and occlusion of saphenous vein grafts [29]. Although the use of BITA increases long-term survival [2, 3], it has also been associated with increased risk of DWSI and mediastinitis [30].
Saphenous Vein Grafts Display Poor Endothelium-Dependent and Endothelium-Independent Dilation-Implications for the Pathogenesis of Vein Graft Atherosclerosis
2008, Heart Lung and CirculationCitation Excerpt :In addition, direct comparisons of vein graft endothelial function before and after implantation have shown a significant loss of endothelium-dependent dilation.7,9 Poor endothelial function of SVG has also been shown through comparison with internal mammary arteries (IMA)8 and this seemed to be more pronounced in diabetic patients.14 These findings are consistent with the poor reactivity seen in our study in vivo.
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